Winstrol Review

October 5, 2007 | Leave a Comment

Winstrol is the common commercial brand of the anabolic steroid called stanozolol. It is probably one of the most used anabolic steroids worldwide. It became notorious due it was the cause for the positive anti-doping test of Ben Johnson. Ben was then the 100 meters world record holder and Olympic gold medallist.

You can find a complete winstrol – stanozolol profile at:


http://www.winstrol.info/1/winstrol-profile/

As I stated before winstrol is the well known brand of stanozolol, but in the recent years another brand from British Dragon has become notorious as well. This pharmaceutical laboratory carries a wide range of anabolic steroids of the highest purity.

British Dragon also implemented very strict procedures for authenticating and establish as genuine the different distributors they approved. You can check it out at:


http://www.britishdragon.com/sellers/index.php

Probably two of the most know British Dragon products containing stanozolol are:

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Recent medical studies show that stanozolol steroid (aka Winstrol) inhibits leptin secretion in females, but not in male samples of omental adipose tissue in vitro.

Leptin is the product of the product of the obesity gene. Leptin is a 16 kDa protein hormone that plays a key role in regulating energy intake and energy expenditure, including the regulation (decrease) of appetite and metabolism. Leptin is also an adiposity signal. To date, only leptin and insulin fulfill the criteria of an adiposity signal:

Due it circulates at levels proportional to body fat.
Because it enters the central nervous system (CNS) in proportion to its plasma concentration.
Due its receptors are found in brain neurons involved in regulating energy intake and expenditure.

Study of Journal of Endocrinology, Vol 160, Issue 3, 425-432 showed that circulating leptin levels are higher in women than in men, even after correction for body fat. This gender-based difference may be conditioned by differences in the levels of androgenic hormones. To explore this possibility, a systematic in vitro study with organ cultures from human omental adipose tissue, either stimulated or not with androgens (1 microM), was undertaken in samples obtained from surgery on 44 non-obese donors (21 women and 23 men). The assay was standardized in periods of 24 h, ending at 96 h, with no apparent tissue damage. Leptin results are expressed as the mean+/-s.e.m. of the integrated secretion into the medium, expressed as ng leptin/g tissue per 48 h. Spontaneous leptin secretion in samples from female donors (4149+/-301) was significantly higher (P<0.01) than that from male donors (2456+/-428). Testosterone did not exert any significant effect on in vitro leptin secretion in either gender (4856+/-366 in women, 3322+/-505 in men). Coincubation of adipose tissue with dihydrotestosterone (DHT) induced a significant (P<0.05) leptin decrease in samples taken from women (3119+/-322) but not in those taken from men (2042+/-430). Stanozolol, a non-aromatizable androgen, decreased (P<0.05) leptin secretion in female samples (2809+/-383) but not in male (1553+/-671). Dehydroepiandrosterone sulphate (DHEA-S) induced a significant (P<0.01) leptin decrease in female samples (2996+/-473), with no modifications in samples derived from males (1596+/-528). Exposure to androstenedione also resulted in a significant reduction (P<0.01) of leptin secretion in samples taken from women (2231+/-264), with no effect on male adipose tissue (1605+/-544). In conclusion, DHT, stanozolol, DHEA-S and androstenedione induced a significant inhibition of in vitro leptin secretion in samples from female donors, without affecting the secretion in samples from men. Testosterone was devoid of activity in either gender.

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  • Recommended Winstrol - Stanozolol Source


    Steroid Pharmacy

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    Anyway there are few excellent pharmaceutical sources. You can check a totally legit pharmaceutical source out at:

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